Ethyl Trans-4-oxo-2-butenoate For Minodronic Acid

Ethyl Trans-4-oxo-2-butenoate For Minodronic Acid

Intermediate of Minodronic acid .
Indications of Minodronic acid : Osteoporosis
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Product Details

Chemical Name

Ethyl trans-4-oxo-2-butenoate CAS 2960-66-9

Synonyms

Ethyl   fumaraldehydate; 
(E)-Ethyl 4-oxobut-2-enoate
(2E)-4-Oxo-2-butenoic Acid Ethyl Ester; 

Fumaraldehydic Acid Ethyl Ester; 

(2E)-4-Oxobut-2-enoic Acid Ethyl Ester; 
(E)-Ethyl 4-Oxo-2-butenoate; 
Ethyl (2E)-4-Oxobut-2-enoate; 

Ethyl Fumaraldehydate; 
Ethyl trans-4-Oxo-2-butenoate;

CAS   No.

2960-66-9

Description  

Osteoporosis is a bone disease characterized by low bone mass and structural degradation of bone tissue. Osteoporosis treatments currently used clinically, such as estrogen, selective estrogen modulators, calcitonin, and bisphosphonates, are mainly beneficial to reduce bone resorption; there are also some treatments such as fluoride and Parathyroid hormone can increase bone formation. Anti-bone resorption therapy is very effective in treating osteoporosis, even if it usually does not induce the formation of new bone.

Anti-bone resorption therapy is very effective in treating osteoporosis, even if it usually does not induce the formation of new bone. To date, bisphosphonates are still the most widely used anti-bone resorption drugs.They are all synthetic analogs of endogenous mineral deposition   inhibitor pyrophosphate, which can inhibit bone resorption and increase bone mineral density ( BMD) can effectively treat osteoporosis, Paget's disease and tumor-related bone disease, but its mechanism of action is not clear.

Ethyl Trans-4-oxo-2-butenoate CAS 2960-66-9 is the mainly   intermediate of Minodronic acid.

Minodronic acid: Osteoporosis

Administration of minophosphonic acid can effectively   inhibit bone loss and bone microstructure degradation caused by inflammation. This product can significantly inhibit the increase in the number of   osteoclasts and bone resorption caused by arthritis and the acceleration of bone turnover, and can inhibit the reticulation Bone loss and maintenance of bone microstructure.


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